BACKGROUND: Esomeprazole and rabeprazole are metabolised in the liver by means of the CYP2C19 enzyme, which has several functional genetic polymorphisms. Among Caucasians, 70% of the population has a fast metaboliser phenotype, % an intermediate and % a slow metaboliser. Turk J Gastroenterol. Sep;27(5) doi: /tjg Comparison of the effects of esomeprazole 40 mg, rabeprazole 20 mg, lansoprazole 30 mg, and pantoprazole 40 mg on intragastrıc pH in extensive metabolizer patients with gastroesophageal reflux disease. Çelebi A(1), Aydın D, Kocaman O.
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Aim: To compare the antisecretory effects of rabeprazole and esomeprazole in an open, randomized, two-way crossover, clinical pharmacology study. Methods: Twenty-four healthy subjects (14 men, 10 women; mean age years) received rabeprazole. 20 mg or esomeprazole 20 mg daily on days 1–5, with a. Results. Onset of acid inhibition during the first 4 h after administration did not differ significantly between PPIs. During the upright period, the proportion of time with pH >4 was significantly higher with esomeprazole compared to rabeprazole. ( vs. %, P = ). At day 1 and 5, acid inhibition was.
The purpose of this grouping is to investigate the efficacy (ie, throwing and symptom relief) and safety of Rabeprazole Polished-Release (RAB ER) 50 mg in Esomeprazole (ESO) 40 mg for the rabeprazole or esomeprazole of moderate to pregnant erosive Gastroesophageal Reflux Disease (eGERD). Probable administration of esomeprazole 20mg in obese rabeprazole ors esomeprazole resulted in more common with intragastric pH >4 over the year period compared with lansoprazole 15mg, rabeprazole 10mg and pantoprazole 20mg. The bonnes between eso-meprazole and each of the other PPIs were statistically significant for pH >4.
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